HYPERPARATHYROIDISM

Authors

  • Nazir Ahmed
  • Syed Sultan
  • A. Karim Saeed

Abstract

Excessive secretion of Parathyroid Hormone may be primary due to adenoma or
hyperplasia of the parathyroid glands, secondary due to persistent hypocalcaemia or
tertiary when secondary hyperplasia leads to autonomous over activity of the glands.
Whatever the cause increase PTH production leads to hypercalcaemia. This may be
symptomless or may present with bizarre symptoms such as psychosis, renal stones,
anorexia, abdominal pain, polyurea, osteoporosis, pathological fractures and deformities.

References

Adams, P.H., Josey, J., Kelly, P.J., L., Kinney, V.R. and Jones, J.D. Quarterly Journal of Medicine

; 36:1.

Fisken, R.A., Heath, D.A. and Bold, A.M. Quarterly Journal of Medicine 1980; 49:405.

Lowe, L.E., Bird, Ed and Thomas, W.C. Journal of Clinical Endocrinology and Metabolism 1962;

:261.

Mayer, W.P.L. Archives of Surgery 1960; 80:308.

Roberts, W.C. and Waller, B.F. American Journal of Medicine 1981; 71:371.

Stanbury, S.W. and Lumb, G.A. Quarterly Journal of Medicine 1966; 35 :1.

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How to Cite

Ahmed, N., Sultan, S., & Saeed, A. K. (1988). HYPERPARATHYROIDISM. Journal of Ayub Medical College Abbottabad, 1(3), 29–31. Retrieved from https://jamc.ayubmed.edu.pk/index.php/jamc/article/view/4628