AETIOLOGY OF TRICUSPID REGURGITATION
AbstractBackground: Tricuspid regurgitation (TR) is regarded as a secondary disorder. Aim of the study wasto know what percentage is secondary to heart and lung disease and its prevalence in normal adults.Methods: Two hundred and 30 adults with clinically detectable TR were studied clinically to know thecause of TR. Results: Thirteen percent of the adults were normal without any detectable cause for TR.In others, 24% of TR cases were secondary to ischemic heart disease (IHD) and hypertension wasfound in 14% cases. Sixteen percent had rheumatic heart disease (RHD) while chronic obstructive lungdisease was found in 23% cases. The rest of 10% cases of TR had cardiomyopathy (CMP) andcongenital heart disease as secondary causes. Conclusion: Ischemic heart disease, COPD andhypertension are common causes of TR. Others include RHD, CMP and congenital heart disease.Thirteen percent of apparently normal adults had TR.Keywords: Tricuspid regurgitation, IHD, Hypertension, COPD
Karius KB, Klaster EF, Bristol JD, Lees MH, Griswold HE.
Problems in hemodynamic diagnosis of Tricuspid Insuffiency.
Am Hear J 1988;75:173–9.
Hansung CE, Rowe GG. Tricuspid incompetence- A study of
hemodynamics and pathogenesis. Circulation 1972;45:793–9.
Shah PM. Tricuspid valve, prosthetic valve and multi-valvular
heart disease. In Hurst`s The Heart, 12th edition New York:
McGraw Hill; 2008. p. 1770–80.
Waller BF, Moriarty AT, Eble JN, Daavey DM, Hawely DA,
Pless JE. Etiology of tricuspid regurgitation based on annular
circumference leaflet area in analysis of 45 necropsy patients with
clinical and morphological evidence of pure Tricuspid
regurgitation. J Am Coll Cardiol 1986;7:1063–72.
Gordon A Ewy. Tricuspid Valve disease. In: Alpert JS, Dalen JE,
Rahimtoola SH (eds). Valvular heart disease. 3rd edition
Philedelphia: Lippincott Williams & Wilkins 2000.p 377–89.
Muller O, Shillingford J. Tricuspid Incompetence. Br Heart J
Tei C, Shah PM, Cherian G, Trim PA, Wong M, Ormiston JA.
Echocardiographic evaluation of normal and prolapsed tricuspid
valve leaflets. Am J Cardiol 1983;52:796–800.
Feigenbaum H, Armstrong WF, Ryan T. Tricuspid and pulmonary
valve. In: Feigenbaum’s Echocardiography, 7th edition.
Philadelphia: Lippincott Willium & Wilkins 2008:346–68.
Cha SD, Gooch A. Diagnosis of tricuspid regurgitation. Current
status. Arch Intern Med 1983;143:1763–8.
Bonow RO, Carabello BA, Kanu C, de Leon AC Jr, Faxon
DP, Freed MD, et al. ACC/AHA Task force on practical
guidelines. ACC/AHA 2006 guidelines for the management of
patients with valvular heart disease: a report of the American
College of Cardiology/American Heart Association Task Force on
Practice Guidelines (writing committee to revise the 1998
Guidelines for the Management of Patients With Valvular Heart
Disease): developed in collaboration with the Society of
Cardiovascular Anesthesiologists: endorsed by the Society for
Cardiovascular Angiography and Interventions and the Society of
Thoracic Surgeons. Circulation 2006;114(5):e84–231.
Yoshida K, Yoshikawa J, Shakudo M, Akasaka T, Jyo Y, Takao
S, et al. Color Doppler evaluation of valvular regurgitation in
normal subjects. Circulation 1988;78(4):840–7.
Keller CA, Shepard JW Jr, Chun DS, Vasquez P, Dolan GF.
Pulmonary hypertension in chronic obstructive pulmonary disease.
Multivariate analysis. Chest 1986;90(2):185–92.
Irwin RB, Luckie M, Khattar RS. Tricuspid regurgitation:
contemporary management of a neglected valvular lesion.
Postgrad Med J 2010;86(1021):648–55.
Sagie A, Sshwammenthal E, Newell JB, Harrell L, Joziatis
TB, Weyman AE, et al. Significant Tricuspid regurgitation is a
marker for adverse outcome in-patients undergoing percutaneous
balloon mitral valvuloplasty. J Am Coll Cardiol 1994;24:696–702.
Missri J, Agnarsson U, Sverrisson J. The clinical spectrum of
tricuspid regurgitation detected by pulsed Doppler
echocardiography. Angiology 1985;36(10):746–53.
Behm CZ, Nath J, Foster E. Clinical correlates and mortality of
hemodynamically significant tricuspid regurgitation. J Heart Valve
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